A few about seborrheic keratosis

Seborrheic keratosis is a benign tumor that never become cancer. Actually the most danger is that it is very annoying. Seborrheic keratosis tumors come in different shapes and sizes, from large black spots faint dots.

Characteristics of seborrheic keratoses

The wicked witch with a wart on the nose probably had a seborrheic keratosis. How can you tell if that bump on the face or chest is actually a seborrheic keratosis? Conditions for seborrheic keratoses have some specific features:

  •     They look to be “glued” to the skin. Are classically described a drop of clay or mud stuck on the skin. The edge of the seborrheic keratosis is not connected to the underlying skin so it looks like it could be removed with a fingernail.
  •     Warty surface – Seborrheic keratoses may seem. As some of its development can have very rough surface with a deep fissures that looks like a cauliflower.
  •     Smooth pearly – some seborrheic keratoses are not rough. On the contrary, are smooth, with small bumps somewhat darker than the surrounding tissue.
  •     Itching – some seborrheic keratoses tend to bite especially with advancing age. Some people try to “remove” a seborrheic keratosis and unwittingly make it even more irritated. If irritated enough, the skin around the lump may redden and seborrheic keratoses can bleed. In this case you should alarm you and make an appointment with a doctor immediately.

What treatment has seborrheic keratoses?

The first and usually the best option is to leave it alone. You can get bigger, but still not a precancerous training, so you can keep your skin in a lifetime without a major problem. If the spots are removed, it is usually because they bite, they interfere with clothing or jewelry, or that are aesthetically unacceptable.

Removal of seborrheic keratosis

If you decide to remove a seborrheic keratosis, there are several ways to do it:

  • Liquid nitrogen. A small seborrheic keratosis may be frozen with liquid nitrogen. Liquid nitrogen is intended to freeze and destroy the cells of seborrheic keratoses, leaving the connective tissue intact. After the operation is a scab. When it falls off after several days, the skin underneath will have already begun to repair itself. Removal with liquid nitrogen can leave a scar on the skin. The scar is generally flat, at least you have a tendency to form keloids.
  • Curettage. Another way to remove seborrheic keratoses is amputated. After the operation is usually applied a chemical agent such as aluminum chloride or silver nitrate to stop bleeding. Silver nitrate is a dark brown color and can stain the wound. This color usually disappears after repair of the skin, but in some cases may remain pigmentation. For this reason, the silver nitrate is not used in the face.

Sometimes, seborrheic keratosis can be very difficult to distinguish from melanoma: irregular edges and color variation in its surface. You should not hesitate to consult your doctor about any rash or bumps that concern you.


What is Seborrheic Keratoses?

1 – Seborrheic keratoses
Seborrheic keratoses or seborrheic warts are common benign skin lesions, especially in the elderly. They belong to the category of benign epithelial tumors and those are the most common of them.
Single or multiple, they save mucous membranes, palms and soles and are happy in the seborrheic areas of the face, neck or back. They appear most often around the age of 40 years but can occur in adolescence in men as in women.

There are different clinical forms of these lesions (see http://seborrheic-keratosis.com for pictures). They are usually pigmented, although limited, as placed on the skin as macule, papule or plaque with the stage of development, sometimes pedunculated, light brown in color (from yellow to dark brown). Their warty surface is progressively covered with a coating squamocolumnar keratotic fat. Lesion size varies from a few millimeters to several centimeters but rarely exceeds 1 cm.

When multiple, they can be distributed along skin folds, lines Blashko or Christmas tree.

Some lesions may be inflammatory due to the rupture of pseudocysts they contain or secondary to trauma or, more rarely due to infection.

The course is chronic and benign. These lesions are frequently asymptomatic but may become sensitive or itchy, erythematous or crusting, including the support of trauma.

Diagnostic tools: Histologically, they are composed of epidermal hyperplasia with strong predominance of basal cells. Hyperplasia is separated by invaginations of the stratum corneum forming cystic cavities or “horny pseudocysts” characteristics. Hyperpigmentation is common. There are different histological types of seborrheic keratosis: acantholytic, hyperkeratotic, reticulated or adenoid, irritated or clonal based on the preponderance of acanthosis, papillomatosis or of hyperkeratosis. The presence of atypical mitoses moderate or must discuss a local irritation or inflammation. In this case, there is often a dermal lymphocytic infiltrate, perivascular or diffuse lichenoid.

Most often, the diagnosis is easy and clinic.
Dermoscopy can be useful to confirm the clinical diagnosis. The analysis will identify dermatoscope it is not melanocytic lesion (without pigment network without globule, streak boss homogeneous and parallel). It shows the presence of pseudocysts horny as round structures of variable size, white or yellowish. Can also be observed pseudo-comedo, structures circumscribed, irregularly shaped, yellow-brown to brown-black, corresponding to keratin plugs within dilated follicular orifices.

Seborrheic keratoses and tumors: The seborrheic keratoses can coexist with other benign or malignant tumor (collision). The sign of Leser-Trelat is the sudden efflorescence of multiple pruritic seborrheic keratoses nature. This sign directs to the presence of a solid tumor, most commonly a gastric cancer or adenocarcinoma, rarely lymphoma or breast cancer or melanoma.

The etiology of these tumors is unknown. Sun exposure, the genetic predisposition have been implicated as possible contributing factors. There is often a family history identical, these lesions frequently appear in the photo-exposed areas. The role of viral infection with HPV has also been raised, particularly because of the clinical appearance of warts close. If the association of HPV with genital seborrheic keratoses has been shown in one study, it does not seem confirmed for seborrheic keratoses extragenital.

Differential diagnoses to consider are: achrocordon, common warts, genital warts, the acrokeratosis verruciformis, tumors of the follicular infundibulum, the eccrine poromes, Bowen’s disease, squamous cell carcinoma, lentigo, nevus or melanoma.

The papular dermatosis stuccokératose and black variants are seborrheic keratoses in hyperkeratotic forms for the first and for the second acantholytic, demonstrated by the presence of somatic mutations (in these lesions) genes PIK3CA and FGFR3 like keratoses Seborrheic classics.
The treatment is most often dictated by aesthetic constraints. Symptomatic lesions can be treated by curettage or cryotherapy. Surgical treatment (resection) is recommended in cases of diagnostic uncertainty, especially with melanocytic tumor, where rapid growth or atypical features.
The inverted follicular keratosis or eccrine porome is a benign epithelial tumor, considered by some authors as a follicular tumor. This is the main differential diagnosis of seborrheic keratosis irritated.

This lesion is rarer than seborrheic keratosis, usually occurs in adults, middle age, after 60 years. Men are more frequently affected than women (2/1).

Clinically: This is a single lesion, firm, white or flesh-colored or pink, usually located on the face (cheeks, lips above). The size of the lesion is variable, often less than 1 cm in diameter but up to ten centimeters.

Histology: It is endophytic or exophytic lesions, often symmetrical and well circumscribed. The lesion consists of a proliferation of keratinocyte medium or large, distinct from each other with multiple elements dyskeratosis, hyperkeratosis topped ortho and para-keratosis. Basaloid cells can be observed in the periphery.

The discovery of inverted follicular keratoses can discuss Cowden syndrome because it is one of the minor criteria of this disease. The link between the follicular keratosis inverted the trichilemmome, seborrheic keratosis and verruca vulgaris is discussed.

The treatment is surgical.

Seborrheic Keratosis – Causes, Symptoms and Remedies

Clinically known to be seborrheic or seborrheic Keratoses, these are nothing but spots that are developed in one’s body due to old age. Another very common name for this disease is senile warts and to be specifically correct senile warts are harmless skin lesions developed in an individual’s body during adult age. Some other names for this disease include basal cell papillomas and brown warts.

To begin with, we must look at the symptoms of seborrhoeic Keratoses or senile warts as it is generally known. These spots first appear in a person body like tanned skin but the difference is that they appear in light spots. Initially, they are flattened but over time the color of these spots darken and a brown more thick skin overtone is developed. Furthermore, there is no physical feeling to the individual of senile warts. They are predominantly dead skin and the part covering the body is usually not noticeable of any pain. In some cases, the senile warts develop a pinkish texture while more commonly it is either dark brown or black in color. All in all, one must know no matter what the color is senile warts are completely harmless.

The cause of senile warts is not accurately known to man up till now, but it is a general clinical perception that senile warts are the direct consequences of aging. Since they are predominantly harmless, senile warts are usually degenerative in nature but some argue that prolonged exposure to ultraviolet light may accelerate the generation of senile warts in an individual.

The remedies of senile warts can be put into three main categories, the first and the most common way of removing senile warts is through laser therapy. This is more of a permanent fix to developing senile warts and since the success rate and dangers of the procedure are both minimum therefore, more and more people are opting to go for the laser therapy to reduce senile warts.

Cyrotherapy and Curettage are the other two common methods to reduce warts. In cyrotherapy, the patient is kept under a series of varying temperatures usually cold temperature and this helps in reducing the senile warts present in the individual’s body. It is however a very slow process, just like physiotherapy, and it takes a while to get the warts to disappear from the body.

Curettage is another very effective way to diminish senile warts and in the previous two decades this method was the most popular one of the three. With laser therapy coming into thescene people are moving away from Curettage and opting for laser treatment. In Curettage the tissues from the upper skin of body is removed and consequently the senile wart present are also diminished. Similarly this is done all over the body until the senile warts are reduced entirely but the problem is that with curettage and cyrotherapy senile warts do not tend to be reduced permanently and there is chance or reappearance as well.